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Fungal Infections | Candida | Pathology Video Lectures | V-Learning |

Working towards delivering best pathology lectures on, here is a lecture on fungal infections. Starting with fungal pneumonia, candidal infections pathogenesis i.e. candidiasis, and pulmonary aspergillosis morphology in conjunction with allergic bronchopulmonary aspergillosis diagnostic criteria comes under debate. Subsequently, light is shed on cryptococcosis, acute self-limited histoplasmosis and disseminated histoplasmosis.

Lecture Duration – 00:57:06
Release Date – June 2019

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Morphological forms of fungi encompass yeasts and molds. After that information about various stains is delivered that are used to detect fungal infections. One of the fungal infections, Pneumocystis Jiroveci Pneumonia, discussed over here causes pneumonia in people with impaired cell- mediated immunity. Its epidemiology, pathophysiology, and pathology are discussed subsequently. Clinical features include fever, shortness of breath, nonproductive cough and dyspnea.

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Prime emphasis of this section is Candida. Maheen Farooq notifies that candidal infections of deep tissues can be life-threatening. However, they are less common. Talking about the pathophysiology, educator tells that common causative factor for candidiasis is the suppression of competing bacterial flora by the use of antibiotics. Similarly, comments are given on pathology. Thrush, candidal vulvovaginitis, and candidal endocarditis are some of the clinical features of candidal infections.

As aspergillus species are the common environmental fungi, Maheen Farooq talks about the three types of pulmonary aspergillosis in this section. Allergic bronchopulmonary aspergillosis results by the inhalation of aspergillus spores. The most frequent human pathogen, Aspergillus fumigatus morphology, is also discussed. Moreover, pathology and clinical features are delineated. Coming towards the elaboration of treatment modality, surgical excision is indicated in some cases.

Cryptococcosis pathogenesis mainly affect Meninges and lungs. Talking about the pathophysiology, immunologically intact individuals can kill C. neoformans with the help of neutrophils and alveolar macrophages while in individuals with defective cell-mediated immunity, the cryptococci survive. Later on, pathology is pursued. Also, cryptococcal meningoencephalitis is discussed along with clinical features.

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Maheen Farooq elaborates histoplasma capsulatum which is a dimorphic fungus. After that pathophysiology of histoplasmosis is highlighted whose primary infection begins with phagocytosis of microconidia. Pathologically, acute self-limited histoplasmosis Is characterized by necrotizing granulomas. Following this, information about disseminated histoplasmosis is delivered in conjunction with clinical features.


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